Source: Clinical Endocrinology. Unidade: FM
Assunto: ENDOCRINOLOGIA
ABNT
JORGE, Alexander A. L. et al. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration. Clinical Endocrinology, v. 60, p. 36-40, 2004Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2004.01930.x. Acesso em: 02 maio 2024.APA
Jorge, A. A. L., Souza, S. C. A. L., Arnhold, I. J. P., & Mendonça, B. B. (2004). The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration. Clinical Endocrinology, 60, 36-40. doi:10.1111/j.1365-2265.2004.01930.xNLM
Jorge AAL, Souza SCAL, Arnhold IJP, Mendonça BB. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration [Internet]. Clinical Endocrinology. 2004 ; 60 36-40.[citado 2024 maio 02 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.01930.xVancouver
Jorge AAL, Souza SCAL, Arnhold IJP, Mendonça BB. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration [Internet]. Clinical Endocrinology. 2004 ; 60 36-40.[citado 2024 maio 02 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.01930.x